Chemiluminescent Solution（Allergy）
Series	Product Name	Product Name
Allergen-specific IgE	House dust mite D1	Ribwort W9
	Dust mite D2	Kentucky blue grass G8
	Cat dander E1	Cultivated rye G12
	Dog dander E5	Pistachio F203
	Sesame seed F10	Maple leaf sycamore, London plane, Plane tree T11
	Peanut F13	Latex K82
	Soybean F14	Olive T9
	Milk F2	Strawberry F44
	Crab F23	Cypress T23
	Shrimp F24	Almond F20
	Egg F245	cross-reactive carbohydrate determinants CCD
	Beef F27	White Ash T15
	Codfish F3	Apple F49
	Wheat F4	Cladosporium herbarum M2
	Mutton F88	Penicilloyl G C1
	House dust H1	Penicilloyl V C2
	Cockroach, German I6	Amoxicilloyl C6
	Aspergillus fumigatus M3	Guinea pig epithelium E6
	Alternaria M6	Rye F5
	Willow T12	Rice F9
	Common ragweed W1	Tomato F25
	Mugwort W6	Pork F26
	Cocksfoot G3	Carrot F31
	Common silver birch T3	Potato F35
	Hazel T4	Yeast F45
	Egg white F1	Egg yolk F75
	Timothy grass G6	Gluten F79
	Garlic F47	Peach F95
	Kiwi F84	Apricot F237
	Celery F85	Walnut F256
	Mustard F89	Bermuda grass G2
	Banana F92	Johnson grass G10
	Oak T7	Velvet grass G13
	Goosefoot W10	Bahia grass G17
	Hazel nut F17	Honey bee venom I1
	Horse dander E3	Yellow jacket venom I3
	Rabbit epithelium E82	Paper wasp venom I4
	Hamster epithelium E84	Penicillium chrysogenum M1
	Meadow fescue G4	Grey alder T2
	Rye-grass G5	Mountain juniper T6
	Cultivated wheat G15	Eastern Wall pellitory W19
	Beech T5	Spreading pellitory W21
	European ash T25	Japanese Hop W22
	Dandelion W8	/

Allergic diseases are conditions in which a patient inhales or ingests substances containing allergenic components (called allergens or allergens, allergen) that trigger the body's B cells to produce excessive immunoglobulin E (IgE). When IgE antibodies are re-exposed to allergens in vivo, they cross-link with allergens and bind to the higher affinity receptor FcεRI on the surface of mast cells and basophils, resulting in FcεRI accumulation and mast cell and basophil activation. During activation, mast cells degranulate and release histamine, an inflammatory mediator stored in cytoplasmic granules, and leukotrienes, immunoreactive prostaglandins, cytokines and chemokines such as IL-4 and IL-5 synthesized through arachidonic acid pathway, triggering disease symptoms of allergic reactions (or allergic reactions), such as allergic asthma, hay fever, urticaria, allergic rhinitis, eczema, allergic dermatitis, conjunctivitis and gastrointestinal dysfunction. The detection of different kinds of allergen-specific IgE antibodies in serum can be used for auxiliary diagnosis of whether some symptoms are caused by allergy in clinical practice. Immunoglobulin IgE is an antibody that mediates type I allergic reactions, and allergen-specific IgE is present in the serum of allergic patients, which is called specific IgE. Those allergic to milk have IgE against milk allergens; those allergic to artemisinin pollen have IgE against the pollen. Allergens enter the body to induce the production of specific IgE, which binds to mast cells and basophils, allowing the body to enter a state of specific sensitization to this allergen. When the allergen comes into contact again, it binds to the IgE receptor on the cell membrane and causes a series of biochemical reactions, which subsequently release various bioactive mediators related to allergic reactions and inflammation, such as histamine. Since this antibody can only specifically bind to this allergen, it is necessary to use purified allergen instead of anti-IgE for detection. There are many specific IgE assay methods, such as ELISA, FEIA, and immunoblotting are commonly used in clinical practice.